The Only Blood Serum Mycotoxin Test in the U.S.
Test for 14 mycotoxins with both IgG and IgE antibody detection. Total 28 results that show how your immune system is responding to mycotoxin exposure.
Mycotoxins are invisible toxic compounds produced by certain molds. Exposure can happen through water-damaged buildings, and workplace environments — often without any visible signs of mold. Because mycotoxin symptoms like fatigue, brain fog, headaches, and gut issues overlap with conditions like Chronic Fatigue Syndrome, Fibromyalgia, and Chronic Lyme disease, they’re frequently misdiagnosed or missed entirely. If no one has tested your blood for mycotoxin antibodies, your evaluation may not be complete.
Test Kit Includes: a blood draw tube, a serum sample tube, a test tube mailer box, instructions and a pre-paid return envelope (U.S. patients only)
Price: $380.00 including U.S. shipping
Blood Serum Testing · 14 Mycotoxins · 28 Results · IgG + IgE Antibodies · Research-Backed
Why Blood Serum Testing Matters
Most mycotoxin testing in the U.S. relies on urine samples. MyMycoLab tests blood serum and that difference changes what your results can tell you.
Urine testing detects mycotoxin metabolites, not the actual mycotoxins themselves. Low levels of mycotoxins are naturally present in everyday foods like coffee, grains, and nuts. Meaning urine results can reflect what you ate, not whether your body is being harmed. Blood serum testing measures your immune system’s actual response: the IgG and IgE antibodies your body produces when it recognizes mycotoxins as a threat.
Blood Serum Testing
Urine Testing (Other Labs)
What it measures
IgG and IgE antibodies — your immune system's response to mycotoxin exposure
Free mycotoxin metabolites — the toxin compounds passing through your body
What results tell you
Whether your body has mounted an immune reaction to specific mycotoxins — indicating meaningful exposure
Whether mycotoxin compounds are present in your urine at the time of collection
Ochratoxin A detection
Reliable. Detected through antibodies in the blood
Unreliable. 99.8% of ochratoxin binds to albumin and is reabsorbed by the kidneys, making urinary excretion negligible
Dietary mycotoxin interference
Not a factor. Antibodies reflect immune response, not food intake
Can be misleading. Can detect traces from everyday foods (cereals, coffee, nuts, wine), even in healthy individuals
Exposure vs. presence
Distinguishes between real immune exposure and incidental contact
Detects presence but cannot differentiate between dietary intake and environmental exposure
Antibody types
Tests both IgG (ongoing/past exposure) and IgE (allergic sensitization) for each mycotoxin
Not applicable. Urine testing does not measure immune response
Number of mycotoxins tested
14 mycotoxins with both IgG and IgE — delivering 28 individual results
Typically, 11–16 mycotoxins, varies by laboratory
Clinical validation
Serum antibody methodology validated in peer-reviewed research for over 20 years
Methodology varies by lab; the CDC has noted limitations of urine-based mycotoxin testing for clinical diagnosis
14 Mycotoxins.28 Results.
Our blood serum panel tests for IgG and IgE antibodies to each of the 14 mycotoxins below — giving you 28 individual data points that reveal how your immune system is responding to mycotoxin exposure.
Every mycotoxin tells a different story about what’s happening in your body. Our panel tests for all 14 — with both IgG and IgE antibodies — so nothing gets overlooked.
Produced by Stachybotrys (black mold), satratoxin is one of the most toxic indoor mold toxins known. It damages brain tissue, triggers widespread inflammation, and causes the death of nerve cells in the part of the brain responsible for smell. It has been found in lung fluid samples taken from infants with pulmonary hemorrhage (lung bleeding), and in those infants’ homes. Satratoxin belongs to the macrocyclic trichothecene family, which are among the most potent inhibitors of protein synthesis in humans.
Produced by Fusarium and Aspergillus molds, these toxins are among the most powerful blockers of protein production in human cells. Without proteins, cells cannot function or repair themselves. These compounds cause tremors, immune dysregulation, and inflammation, and are harmful even in very small amounts. Verrucarins have been specifically identified in Stachybotrys-contaminated indoor environments and in the sera of building occupants.
Ochratoxins come from Aspergillus and Penicillium molds and are among the most commonly detected mycotoxins in the indoor environment. Ochratoxin A (OTA) damages the kidneys, liver, brain, and immune system. Ochratoxins can cross the placental barrier. It can cause cancer and birth defects. It has been classified as a possible human carcinogen since 1993. Importantly, long-term low-level exposure to OTA can be more damaging than a single large exposure. OTA is nephrotoxic, hepatotoxic, neurotoxic, and immunotoxic. It is poorly filtered by the kidneys, which is precisely why urine testing drastically underestimates exposure and why blood serum testing is essential for this toxin. OTA builds up in breast milk, which can expose breastfed babies. Breathing in OTA has been documented to cause kidney failure within 24 hours. Emerging research links OTA to Parkinson’s disease and to autism in children
Produced by Fusarium, T-2 toxin is the only mycotoxin ever used as a biological weapon. In the early 1980s, approximately 100,000 people in Southeast Asia were killed or injured by it. It crosses from the bloodstream into the brain, causes oxidative stress (cell damage from unstable molecules), and disrupts energy production inside cells. T-2 toxin is also a potent endocrine disruptor: it lowers testosterone in both men and women and has been linked to infertility and hormonal problems.
Also called DON, vomitoxin is the most common trichothecene found indoors. In the gut, it blocks protein production, causes DNA damage, triggers inflammation, and breaks down the junctions that hold intestinal cells together, a key step in leaky gut. It crosses into the brain, directly affects neurons and support cells, and disrupts hormone signals. It can raise inflammatory markers by up to 15 times and throws off estrogen and testosterone balance.
Cladosporium is one of the most common molds in both indoor and outdoor air. Its spores are a major trigger for asthma and allergies. Beyond respiratory effects, it releases chemicals that are toxic to the nervous system. Cladosporium has been found in breast cancer tumor samples in large-scale cancer research. [13] Because Cladosporium is so ubiquitous, patients with IgE sensitization to its HSP 70 protein can experience chronic, relapsing respiratory and neurological symptoms with no obvious “moldy” environment making diagnosis particularly challenging.
These trichothecenes from Stachybotrys (black mold) affect nearly every organ system. They trigger inflammation and suppress the immune system. In the gut, they block protein production, cause oxidative stress, and can cause internal bleeding. In the nervous system, they cause tremors, headaches, seizures, sleep problems, and can damage the protective coating around nerves (myelin), leading to conditions similar to multiple sclerosis. They also disrupt hormones, lowering testosterone and raising estrogen in both men and women. In a landmark study, researchers tested blood samples from 44 people living in Stachybotrys-contaminated buildings and found trichothecene toxins in the sera of 23 of them — compared to only 1 out of 26 people from uncontaminated buildings. This was among the first studies to directly demonstrate that indoor mold exposure leads to measurable toxin levels in human blood.
Alternaria produces a toxin called alternariol that is directly toxic to cells and can damage or mutate DNA. It dysregulates immune function and triggers oxidative stress, a process where unstable molecules cause widespread cell damage. It also lowers testosterone. Alternariol is specifically genotoxic: it causes DNA strand breaks and chromosomal aberrations, meaning it does not merely damage cells but can alter the genetic instructions those cells carry forward when they divide.
This toxin from Aspergillus breaks down red blood cells and is specifically toxic to neutrophils and macrophages, two of the most important immune system’s infection-fighting cells. It dysregulates immunity broadly. The destruction of neutrophils and macrophages is particularly consequential because these are the cells the body relies on as its first line of defense against new infections. When hemolysin suppresses them, patients become vulnerable to a cascade of secondary infections that further complicate the clinical picture. Aspergillus has been found in greater amounts inside lung cancer tumors.
Produced by Aspergillus versicolor, sterigmatocystin can cause cancer and birth defects. It damages the liver and has been linked to the development of autoimmune diseases by altering how the immune system communicates and which genes get turned on or off. This epigenetic mechanism where sterigmatocystin changes gene expression without altering the DNA sequence itself may explain why some patients develop autoimmune conditions months or years after their initial mold exposure, long after the mold itself has been remediated.
This Penicillium toxin is a powerful immune dysregulator. It works by blocking the growth of immune cells (lymphocytes), broadly weakening the body’s ability to fight infections and control inflammation. Mycophenolic acid inhibits the enzyme inosine monophosphate dehydrogenase, which is critical for lymphocyte proliferation. This is the same mechanism exploited by the prescription immunosuppressant mycophenolate mofetil (CellCept), used to prevent organ transplant rejection — which underscores just how pharmacologically potent this environmental toxin is.
Produced by Aspergillus fumigatus, gliotoxin dysregulates the immune system, damages nerves, and harms DNA. Studies show it causes demyelination of nerves. Research has linked it to multiple sclerosis. It can kill immune cells even at extremely low concentrations. Gliotoxin is active at picomolar concentrations — quantities so small they are nearly undetectable by standard laboratory methods. It induces apoptosis (programmed cell death) in immune cells, effectively disarming the body’s defenses from the inside.
Fumonisins come from Fusarium mold. They block a critical process in fat metabolism, which has been linked to depression that does not respond to standard antidepressants. Specifically, fumonisins inhibit ceramide synthase, disrupting sphingolipid metabolism — a pathway essential for cell membrane integrity and brain signaling. This disruption of brain lipid chemistry may explain why affected patients experience depression that is biologically resistant to conventional serotonin-based treatments. Disrupting sphingolipid metabolism can lead to Alzheimer’s and Parkinson’s disease. Fumonisin contamination of corn along the Texas–Mexico border has been connected to higher rates of neural tube defects in newborns. Fumonisin B1 is classified as a possible human carcinogen. These toxins also damage the mitochondria (the energy-producing parts of cells) and harm the liver and kidneys.
Produced by Fusarium, zearalenone (ZEA) mimics estrogen in the body. It latches onto estrogen receptors and disrupts the hormonal balance. In women, it lowers the chances of a successful pregnancy and reduces milk production. In young girls, it can trigger early puberty. In men, it reduces testosterone and lowers sperm production. ZEA is classified as a non-steroidal estrogen: it is structurally similar enough to human estrogen to bind estrogen receptors, but its effects are disruptive rather than physiological. A recent study found elevated ZEA in blood samples from stillborn and newborn babies with physical malformations and developmental problems. It is classified as a possible carcinogen by IARC.
Getting Tested is Simple
No physician referral required. Order directly and get results delivered to your patient portal.
The Only Blood Serum Mycotoxin Lab in the U.S.
MyMycoLab is the only laboratory in the United States testing blood serum for 14 mycotoxins with both IgG and IgE antibody detection.
20+ Years of Published Research
MyMycoLab’s blood serum methodology is validated by peer-reviewed studies in medical journals spanning over two decades of clinical research.
Trusted by 2,500+ Practitioners
Healthcare providers across the country rely on MyMycoLab’s blood serum testing to help identify mycotoxin exposure in their patients.